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I have been on Gleevec since March 2007 when I was dignosed with Chronic Mylogenous Leukemia. I was sick for about 4 months on this drug with Nausea, leg swelling, pain so bad that I felt crippled by the end of every day and headaches. I had these symptoms even while taking Zofran with the drug. My doctor's at MSKCC told me to hang in there saying I was going to wake up one day soon and feel better than ever. And then after 4 months, they were right. I remember that I woke up one morning, stretched my arms and legs and yawned and saw the sun shining through my bedroom window and I turned to my husband and said "I feel great"! I have pretty much felt great since then and my life has thrived. I am not just a survivor, I am a thriver!
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I've been on Gleevec since September 2007 for Chronic Myelogenous Leukemia. And I can say that I didn't have any problems with iron level, but maybe I am an exception. However I know many more people who had no problems with their iron while being on Gleevec. Before it I was on another medication but it was not effective at all and had serious side effects for me. but my doctor persuaded me that it was the best possible medication in my case and it had helped many people get on lasting remission. However, it caused me to vomit from time to time, besides I had severe leg cramping and that would make me crazy. In the night I needed several blankets to calm down the cramping and fall asleep. Sometimes I spent sleepless nights and felt incredibly weak and passive next day, especially if I vomited before and had absolutely no energy to live the day out. So after 2 months I insisted on switching to another medication and he put me on Gleevec. I consider it to be a lucky decision and I am happy that this medication is so effective for me and causes no additional problems.

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By the 5-year median follow-up of this trial, imatinib mesylate induced complete cytogenetic response in more than 80% of the participants, with the annual rate of progression to accelerated-phase CML or blast crisis dropping from 2% in the first year to less than 1% in the fourth year.

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High-affinity interaction of tyrosine kinase inhibitors with the ABCG2 multidrug transporter.

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Here we compared CNS tissue lacking signs of demyelination from both groups, as the imatinib-treated group at this time-point had not yet developed the disease.

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Kosei Matsue, Masami Takeuchi, Mihoko Koseki, Hidetaka Uryu.

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Analytical Validation and Application of a Targeted Next-Generation Sequencing Mutation-Detection Assay for Use in Treatment Assignment in the NCI-MPACT Trial.

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ABL PCR testing in chronic myelogenous leukemia: molecular diagnosis for targeted cancer therapy and monitoring.

LCH and a small amount of nearby healthy tissue.

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Graft-versus-leukemia effect in hematopoietic stem cell transplantation for pediatric acute lymphoblastic leukemia: significantly lower relapse rate in unrelated transplantations.

Currently, patients with CML have a median survival of 5 or more years.

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Targeting the innate immune system in pediatric inflammatory bowel disease.

Specific therapy for CGD has not been established and resolution of the abnormalities, either endoscopic or histological, has not been documented.

Emanuel Raschi, Valentina Vasina, Maria Grazia Ursino, Giuseppe Boriani, Andrea Martoni, Fabrizio De Ponti.

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The noncardiac causes that are most serious and require immediate treatment are pulmonary embolus, pneumothorax, and aortic dissection.

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ABL-dependent and -independent mechanisms of imatinib mesylate resistance.

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